Overstock Deal Recombinant Human Tnfrsf1b/cd120b Protein (aa 1-268, 196 Met/arg, His Tag)(active) Save 45% + Free Ship [WIf6xVAz]

Recombinant Human TNFRSF1B/CD120b Protein: Active and Potent

Introducing our premium Recombinant Human TNFRSF1B/CD120b Protein, a bioactive recombinant protein engineered for research and diagnostic applications. This highly purified protein is derived from human HEK293 cells and is tagged with a His tag for easy detection and purification. With its bioactivity demonstrated through its ability to inhibit TNF-α mediated cytotoxicity, this protein is an essential tool for studying the role of TNFRSF1B/CD120b in various biological processes.

• High purity: > 95% as determined by reducing SDS-PAGE.
• Bioactivity: Inhibits TNF-α mediated cytotoxicity with an ED50 of 5-40 ng/mL.

Product Properties and Specifications

Our Recombinant Human TNFRSF1B/CD120b Protein offers several key properties and specifications that make it a reliable choice for researchers. The protein is isolated from HEK293 cells, ensuring consistent quality and purity. It has a calculated molecular weight of 27.7 kDa and an observed molecular weight of 43 kDa. Additionally, the protein is endotoxin-free, with < 1.0 EU per μg as determined by the LAL method.

• Storage stability: Stable for up to 12 months at -20 to -80℃ upon lyophilization.
• Endotoxin levels: < 1.0 EU per μg as determined by the LAL method.

Applications and Benefits

The Recombinant Human TNFRSF1B/CD120b Protein is ideal for a variety of research applications, including cellular signaling, apoptosis, and inflammation studies. This protein can be used to study the role of TNFRSF1B/CD120b in protecting neurons from apoptosis and its involvement in metabolic effects of TNF-alpha. The active form of this protein is particularly beneficial for researchers who need to study the complex interactions between TNFRSF1B/CD120b and TNF-alpha.

• Research applications: Cell signaling, apoptosis, and inflammation studies.
• Bioactivity: Demonstrated by inhibiting TNF-α mediated cytotoxicity.